Physiologie | Institut für Physiologie

Grisk O. The sympathetic nervous system in acute kidney injury. Acta Physiol 2019; DOI: 10.1111/apha.13404.
Witte J, Mühlbauer M, Braun D, Steinbach A, Golchert J, Rettig R, Grisk O. Renal soluble guanylate cyclase is downregulated in sunitinib-induced hypertension. J Am Heart Assoc 2018; 7:e009557.

Grisk O, Koenen A, Meissner T, Donner A, Braun D, Steinbach A, Glöckl G, Zimmermann U, Evert K, Evert M, Katsari E, Löhn M, Plettenburg O, Rettig R. Rho kinase inhibition mitigates sunitinib-induced rise in arterial pressure and renal vascular resistance but not increased renal sodium reabsorption. J Hypertens 2014; 32: 2199-2210.

Grisk O, Schluter T, Reimer N, Zimmermann U, Katsari E, Plettenburg O, Löhn M, Wollert H-G, Rettig R. The Rho kinase inhibitor SAR407899 potently inhibits endothelin-1-induced constriction of renal resistance arteries. J Hypertens 2012; 30: 980-989.
Schlüter T, Zimmermann U, Protzel C, Miehe B, Klebingat K-J, Rettig R, Grisk, O. Intrarenal artery superoxide is mainly NADPH oxidase-derived and modulates endothelium-dependent dilation in elderly patients. Cardiovasc Res 2010; 85: 814-824.

Schlüter T, Steinbach AC, Steffen A, Rettig R, Grisk O. Apocynin-induced vasodilation involves Rho kinase inhibition but not NADPH oxidase inhibition. Cardiovasc Res 2008; 80: 271-279

Rettig R, Grisk O. The kidney as a determinant of genetic hypertension. Evidence from renal transplantation studies. Hypertension 2005; 46: 463-468.

Grisk O, Frey BAJ, Uber A, Rettig R. Sympathetic activity in early renal posttransplantation hypertension in rats. Am J Physiol 2000; 279: R1737-R1744.